It is highly likely that the Alzheimer’s results from a complex and interrelated combination of genetic and non-genetic factors.

These so called risk factors influence a person’s risk to developing Alzheimer’s disease. Currently, each of these risk factors is the subject of a great deal of research around the world.

Although cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes, and smoking, are associated with a higher risk of onset and course of Alzheimer’s, statins, which are cholesterol lowering drugs, have not been effective in preventing or improving the course of the disease.

Long-term usage of non-steroidal anti-inflammatory drugs (NSAIDs) is associated with a reduced likelihood of developing AD. Evidence also support the notion that NSAIDs can reduce inflammation related to amyloid plaques.

Hormone replacement therapy, although previously used, may increase the risk of dementia.

Genetic Risk Factors

A person’s genetic make up can directly influence the chances for the onset and development of Alzheimer’s. A person’s genes are inherited from their biological parents and passed along family lines to their biological children.

There are two main types of Alzheimer’s Disease.

Familial Alzheimer’s Disease (FAD): is a very rare form of Alzheimer’s Disease which runs in families. Also known as Early Onset Alzheimer’s or Younger Onset Alzheimer’s.

Sporadic Alzheimer’s Disease: is the most common form of Alzheimer’s Disease, but researchers are still trying to work out how this develops. Also known as Late Onset Alzheimer’s.

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